[1] 王阳, 米树华, 贾淑杰, 等. 心肌缺血再灌注损伤药物研究现状及进展[J]. 中国实用内科杂志, 2013, 32(12):958-960.
[2] Fernández-Jiménez R, García-Prieto J, Sánchez-González J, et al. Pathophysiology underlying the bimodal edema phenomenon after myocardial ischemia/reperfusion[J]. J Am Coll Cardiol, 2015, 66(7):816-828.
[3] Wei B, Li WW, Ji J, et al. The cardioprotective effect of sodium tanshinone IIA sulfonate and the optimizing of therapeutic time window in myocardial ischemia/reperfusion injury in rats[J]. Atherosclerosis, 2014, 235(2):318-327.
[4] 杨文慧, 郭涛, 杨莉, 等. 体外心脏震波对大鼠心肌梗死后心肌凋亡的影响[J]. 中国老年学杂志, 2016, 36(2):265-267.
[5] Huang H, Nace GW, McDonald KA, et al. Hepatocyte‐specific high‐mobility group box 1 deletion worsens the injury in liver ischemia/reperfusion: a role for intracellular high‐mobility group box 1 in cellular protection[J]. Hepatology, 2014, 59(5):1984-1997.
[6] Nakamura Y, Suzuki S, Shimizu T, et al. High mobility group box 1 promotes angiogenesis from bone marrow-derived endothelial progenitor cells after myocardial infarction[J]. J Atheroscler Thromb, 2015, 22(6):570-581.
[7] Hu H, Zhai C, Qian G, et al. Protective effects of tanshinone IIA on myocardial ischemia reperfusion injury by reducing oxidative stress, HMGB1 expression, and inflammatory reaction[J]. Pharm Biol, 2015, 53(12):1752-1758.
[8] Sun N, Wang H, Wang L. Protective effects of ghrelin against oxidative stress, inducible nitric oxide synthase and inflammation in a mouse model of myocardial ischemia/reperfusion injury via the HMGB1 and TLR4/NF-κB pathway[J]. Mol Med Rep, 2016, 14(3):2764-2770.
[9] He YY, Wen Y, Zheng XX, et al. Intramyocardial delivery of HMGB1 by a novel thermosensitive hydrogel attenuates cardiac remodeling and improves cardiac function after myocardial infarction[J]. J Cardiovasc Pharmacol, 2013, 61(4):283-290.
[10] 胡笑容,谢菁,马瑞松,等. miR-451对心肌细胞缺氧再复氧损伤的保护作用及其机制[J]. 山东医药,2017,57(1):1-3.
[11] 张娟. HMGB1在电针内关穴对心肌缺血再灌注损伤保护作用中的机制研究[D]. 上海:上海交通大学, 2015.
[12] Wang YL, Wang CY, Zhang BJ, et al. Shenfu injection suppresses apoptosis by regulation of Bcl-2 and caspase-3 during hypoxia/reoxygenation in neonatal rat cardiomyocytes in vitro[J]. Mol Biol Rep, 2009, 36(2):365-370.
[13] Xie L, Wu Y, Fan Z, et al. Astragalus polysaccharide protects human cardiac microvascular endothelial cells from hypoxia/reoxygenation injury:the role of PI3K/AKT, Bax/Bcl-2 and caspase-3[J]. Mol Med Rep, 2016, 14(1):904-910.
[14] Wang M, Meng X, Yu Y, et al. Elatoside C protects against hypoxia/reoxygenation-induced apoptosis in H9c2 cardiomyocytes through the reduction of endoplasmic reticulum stress partially depending on STAT3 activation[J]. Apoptosis, 2014, 19(12):1727-1735.
[15] Arslan F, Lai RC, Smeets MB, et al. Mesenchymal stem cell-derived exosomes increase ATP levels, decrease oxidative stress and activate PI3K/Akt pathway to enhance myocardial viability and prevent adverse remodeling after myocardial ischemia/reperfusion injury[J]. Stem Cell Res, 2013, 10(3):301-312.
[16] Evran B, Karpuzoglu H, Develi S, et al. Effects of carnosine on prooxidant–antioxidant status in heart tissue, plasma and erythrocytes of rats with isoproterenol-induced myocardial infarction[J]. Pharmacol Rep, 2014, 66(1):81-86.
[17] 陈亚蓝, 王雪青, 王怡雯, 等. 基于HepG2细胞模型研究普洱茶茶色素的抗氧化作用[J]. 食品科学, 2017, 38(1):226-231.